elk_hntr
New member
Tom - The first case of CWD was seen in 1967 in a captive mule deer at the Foothills Wildlife Research Station (operated by the Colorado Dept. of Wildlife) in Ft. Collins and was attributed then by station employees to close confinement of deer to former (scrapie) sheep pasture or to horizontal transmission from sheep allowed into the pens. The shortest known incubation time in deer is 17 months, dating the exposure back to 1965-66 or earlier. Surplus does were released back into the wild after fawning in the facility; the first case in free-ranging wild deer was seen in 1981. Other infected animals were shipped to zoos (Denver, Toronto, Laramie), game farms, and similar research facilities in Colorado and Wyoming.
Let's take a quick moment to applaud the wonderful research work of our Colorado DOW.
Perhaps this is indeed a naturally occurring prion genetic disease or even transmission at winter feeding stations via rendered downer cow protein (ie, a non-UK strain of bovine spongiform encephalopathy) or CWD deer or elk recycled as rendered road kill. CWD deer are commonly observed at feeding stations in Estes Park, Colorado. Does this make CWD man-made? Just a thought.
Here's some more food for thought.
Can you say conflict of interest. A state fish and game department that derives most or all of its salary and program revenue from the sale of game tags jeopardizes this revenue by disclosure of CWD or by safety warnings to hunters. Seems plausible right? This revenue model is applicable to all 11 western states. Also includes departments of agriculture for different reasons. Conflicts of interest can affect the design of monitoring programs, choice of sampling techniques and pathology method, disclosure of results, and non-adherence to the precautionary principle. Diagnosis: public relation releases from the agency equate absence of evidence to evidence of absence. Example: Colorado fish and game officials held a news conference in 1998 stating they would continue to enjoy eating venison from untested deer and elk from epidemic strongholds because it had not been proven to transmit to human, a vacuous reassurance as no study has ever been conducted. What are your thoughts?
Let's take a quick moment to applaud the wonderful research work of our Colorado DOW.
Perhaps this is indeed a naturally occurring prion genetic disease or even transmission at winter feeding stations via rendered downer cow protein (ie, a non-UK strain of bovine spongiform encephalopathy) or CWD deer or elk recycled as rendered road kill. CWD deer are commonly observed at feeding stations in Estes Park, Colorado. Does this make CWD man-made? Just a thought.
Here's some more food for thought.
Can you say conflict of interest. A state fish and game department that derives most or all of its salary and program revenue from the sale of game tags jeopardizes this revenue by disclosure of CWD or by safety warnings to hunters. Seems plausible right? This revenue model is applicable to all 11 western states. Also includes departments of agriculture for different reasons. Conflicts of interest can affect the design of monitoring programs, choice of sampling techniques and pathology method, disclosure of results, and non-adherence to the precautionary principle. Diagnosis: public relation releases from the agency equate absence of evidence to evidence of absence. Example: Colorado fish and game officials held a news conference in 1998 stating they would continue to enjoy eating venison from untested deer and elk from epidemic strongholds because it had not been proven to transmit to human, a vacuous reassurance as no study has ever been conducted. What are your thoughts?